Identifying Thyroid Problems – Part 1

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Have you recently been diagnosed with thyroid underactivity, or do you have thyroid symptoms? First, let’s review some of the signs and symptoms associated with an underactive thyroid gland, which are usually weight gain, depression, a feeling of cold, psychological slowness, heavy periods, dry skin, fragile and dry hair. Then, for an overly active thyroid gland, the signs or symptoms are generally weight loss, stress, anxiety, irritation, hyperactivity, hand tremors, feeling hot, light, or no menstrual periods. The thyroid gland is located under the skin and muscle tissue at the front of the neck, nearly on the particular area where a bow tie may rest. It is brownish red, with the left and right halves generally weighing less than one gram. Your thyroid is a tiny gland that generates hormones that manage the body’s energy levels and metabolic processes.

 

All of these play a role in your metabolic function by controlling body temperature and maintaining energy. It also plays a crucial role in the development of blood cells and muscle and neurological function. Finally, your thyroid gland allows the body to break down harmful toxins. To be able to prevent disease, it is essential to understand it. And the most important thing to know and understand is the root cause of illness. What is the root cause of this disease? What experiences or situations did the person have that ultimately led to the infection of this disease? Once this is understood, it will be easier to prevent this disease from harming the person.

In thyroid disease, it is more important to know the disease’s root cause because thyroid disease symptoms are usually associated with different diseases or conditions. However, the causes of thyroid problems vary depending on the disease. Other thyroid diseases cause some, some are caused by treatment or medication, some are caused by a lack or excess of certain nutrients in the body, and some are hereditary.

Take Hashimoto’s disease or Hashimoto’s thyroids. This is an autoimmune disease in which your immune system attacks the thyroid gland. This disease’s primary cause is unknown, but doctors and scientists identify several factors that may have caused it. The two most common causes of this disease would be a genetic defect and gender. Genes are thought to play a role in developing Hashimoto’s disease, but scientists have not yet identified which gene(s) is (are) predisposed to this disease. Gender is also thought to play a role, as women are more likely to develop this disease than men (pregnancy is also considered an essential factor in developing this disease). Other causes are thought to be iodine deficiency and radiation exposure.

Like Hashimoto’s disease, the underlying causes of Graves’ disease are also unknown. Genes and gender are thought to play a significant role in the grounds of Graves’ disease. Women also have a greater risk of developing this disease than men, especially pregnant women. Stress and infection are other factors that are believed to cause Graves’ disease. Stress and infection can trigger the disease’s onset in people prone to it, but there are no studies that directly link them to the disease’s cause.

It is vital to note these two diseases because they are the two common causes of most, if not all, thyroid problems.

One of the thyroid problems that can be attributed to these two diseases is thyroids. Thyroids is an inflammation of the thyroid gland. The most common cause of inflammation of the thyroid gland is an autoimmune disease. In this disease, the immune system malfunctions, causing antibodies to attack the thyroid gland. Viruses and bacteria can also cause thyroids because some bacteria and viruses attack the thyroid gland directly. Some medications, such as interferon and amiodarone, also cause thyroids because they damage thyroid cells.

Like thyroids, hypothyroidism is also caused by an autoimmune disease, particularly Hashimoto’s disease. The damage the disease causes to the thyroid gland affects its ability to produce hormones.

The second part continues in another blog.

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